GAD 1 / GAD 67
Synthesizing GABA, the major inhibitory neurotransmitter in the central nervous system
General references GAD 1/ GAD 67
Kinney JW, Davis CN, Tabarean I, Conti B, Bartfai T & Behrens MM (2006). A specific role for NR2A-containing NMDA receptors in the maintenance of parvalbumin and GAD67 immunoreactivity in cultured interneurons. Journal of Neuroscience 26: 1604-15.Tamamaki N, Yanagawa Y, Tomioka R, Miyazaki J, Obata K & Kaneko T (2003). Green fluorescent protein expression and colocalization with calretinin, parvalbumin, and somatostatin in the GAD67-GFP knock-in mouse. Journal of Comparative Neurology 467: 60-79.
Kanaani J, Lissin D, Kash SF & Baekkeskov S (1999). The hydrophilic isoform of glutamate decarboxylase, GAD67, is targeted to membranes and nerve terminals independent of dimerization with the hydrophobic membrane-anchored isoform, GAD65. Journal of Biological Chemistry 274: 37200-9.
Kim J, Richter W, Aanstoot HJ, Shi Y, Fu Q, Rajotte R, Warnock G & Baekkeskov S (1993). Differential expression of GAD65 and GAD67 in human, rat, and mouse pancreatic islets. Diabetes 42: 1799-808.
Tillakaratne NJ, Erlander MG, Collard MW, Greif KF & Tobin AJ (1992). Glutamate decarboxylases in nonneural cells of rat testis and oviduct: differential expression of GAD65 and GAD67. Journal of Neurochemistry 58: 618-27.
