c-Fos

A member of the transcription factor complex AP1

General references c-Fos

Klejbor I, Ludkiewicz B, Domaradzka-Pytel B, Wójcik S & Morys J (2005). Open field stress and neurons containing calcium-binding proteins in the piriform cortex of the rat. Journal of Physiology and Pharmacology 56: 223-331.

Hua F, Harrison T, Qin C, Reifsteck A, Ricketts B, Carnel C & Williams CA (2004). c-Fos expression in rat brain stem and spinal cord in response to activation of cardiac ischemia-sensitive afferent neurons and electrostimulatory modulation. American Journal of Physiology. Heart and Circulatory Physiology 287: H2728-38.

Fleischmann A, Hvalby O, Jensen V, Strekalova T, Zacher C, Layer LE, Kvello A, Reschke M, Spanagel R, Sprengel R, Wagner EF & Gass P (2003). Impaired long-term memory and NR2A-type NMDA receptor-dependent synaptic plasticity in mice lacking c-Fos in the CNS. Journal of Neuroscience 23: 9116-22.

Lallemand D, Spyrou G, Yaniv M & Pfarr CM (1997). Variations in Jun and Fos protein expression and AP-1 activity in cycling, resting and stimulated fibroblasts. Oncogene 14: 819-30.

Kovary K & Bravo R (1992). Existence of different Fos/Jun complexes during the G0-to-G1 transition and during exponential growth in mouse fibroblasts: differential role of Fos proteins. Molecular and Cellular Biology 12: 5015-23.

Kovary K & Bravo R (1991). Expression of different Jun and Fos proteins during the G0-to-G1 transition in mouse fibroblasts: in vitro and in vivo associations. Molecular and Cellular Biology 11: 2451-9.

Nakabeppu Y & Nathans D (1991). A naturally occurring truncated form of FosB that inhibits Fos/Jun transcriptional activity. Cell 64: 751-9.

Dobrazanski P, Noguchi T, Kovary K, Rizzo CA, Lazo PS & Bravo R (1991). Both products of the fosB gene, FosB and its short form, FosB/SF, are transcriptional activators in fibroblasts. Molecular and Cellular Biology 11: 5470-8.

Greenberg ME & Ziff EB (1984). Stimulation of 3T3 cells induces transcription of the c-fos proto-oncogene. Nature 311: 433-8.